People living in counties with strong smoke-free policies were 7.9% less likely to be diagnosed with lung cancer compared with those living in counties with no protection.
A class of DUBs known as the ubiquitin-specific-processing proteases are involved in regulating the p53 signaling pathway and have been implicated in tumorigenesis and radiotherapy resistance.
As breast cancer can be asymptomatic or have a series of different symptoms and physical exam findings, standard procedure is hard to define.
A recent study into the relationship between glucose and cancer cell proliferation may be helping to identify a therapeutic route to starve tumors without killing normal cells.
As post-transplant medications are used to reduce the body's immune reaction to a foreign organ, they may also reduce the body's ability to accurately survey for malignant cells.
Cancer Therapy Advisor asked Shaji Kumar, MD, to share his thoughts about where we are now and where we are going regarding promising data and novel approaches for MM treatment.
Continued, deliberate exploration of the molecular pathways involved in different subsets of the disease is crucial to achieve clinical advances in the future.
Cigarettes may favor VHL-intact RCC, but it is unclear whether cigarette smoking selects for more aggressive histology of RCC in a similar way to, for example, the TP53 mutation in lung cancer.
As more rare cancers are identified, the need for rethinking approaches to research will become greater.
Overexpressed CKAP4 was significantly associated with tumor, nodal, and metastasis stages, as well as Fuhrman grades.
Socioeconomic status, including education and income level, appears to be tied to prostate-specific antigen test results and prostate cancer treatment and survival, but the exact nature of these associations is not yet clear.
Although cytogenetic abnormalities that arise during the course of CML are prevalent in later stages of the disease and are associated with a poorer prognosis, as many as 10% to 12% of patients exhibit ACAs at diagnosis.
Targeting tumor and extracellular LPA could help deny tumors their inflammatory habitat and improve the efficacy of radiotherapy, chemotherapy, and possibly immunotherapies.
It is not always clear whether patients who discontinue anti-PD-1 and -CTLA-4 therapy should re-initiate treatment after adverse events are resolved.
The results of this study support previous findings that psychological distress is associated with an increased risk of mortality among patients with lung cancer.
Regimen and Drug Listings
GET FULL LISTINGS OF TREATMENT Regimens and Drug INFORMATION
|Head and Neck Cancer||Regimens||Drugs|
|Renal Cell Carcinoma||Regimens||Drugs|
Cancer Therapy Advisor Articles
- The Warburg Effect: Can Cancer Be Starved of Sugar?
- Does Radiotherapy Lead to An In Situ Breast Cancer Vaccine?
- Proinflammatory Diet and Colorectal Cancer
- Surgery May Be Superior to Stereotactic Body Radiation Therapy for Early Stage NSCLC
- Smoking After Diagnosis Leads to Worse Renal Cell Carcinoma-specific Survival
- Carfilzomib, Lenalidomide, Dexamethasone Prolong Survival in Multiple Myeloma
- Bortezomib-based Induction Improves PFS, OS in High-risk Newly Diagnosed Multiple Myeloma
- Brentuximab Vedotin Plus Nivolumab May Be Effective Salvage Therapy for Hodgkin Lymphoma
- Brentuximab Vedotin Plus Doxorubicin, Vinblastine, and Dacarbazine Prolongs PFS in Hodgkin Lymphoma
- Poor Prognostic Markers Predict for Abemaciclib Benefit in Patients With Breast Cancer